No, it still wouldn’t address the insulin resistance, just the consequences of the β-cell dysfunction. Ideally therapy would address the hyperglycaemia and preserve β-cell functionality.
Currently insulin is prescribed in T2D when two oral agents aren’t effective at controlling hyperglycaemia.
So while potentially applicable, it would not be a solution.
Ah - when I posted the question I was thinking of my mom who was type 2 and needed to take insulin very regularly (and eventually had a continuous glucose monitor patch on her arm at all times)
It does seem like it should help for cases like that, which I wasn’t aware were less common.
No, it still wouldn’t address the insulin resistance, just the consequences of the β-cell dysfunction. Ideally therapy would address the hyperglycaemia and preserve β-cell functionality. Currently insulin is prescribed in T2D when two oral agents aren’t effective at controlling hyperglycaemia. So while potentially applicable, it would not be a solution.
Thank you for clarifying it.
Ah - when I posted the question I was thinking of my mom who was type 2 and needed to take insulin very regularly (and eventually had a continuous glucose monitor patch on her arm at all times)
It does seem like it should help for cases like that, which I wasn’t aware were less common.
Definitely would help, as part of a most probably already complicated therapeutic regime :)